https://ogma.newcastle.edu.au/vital/access/ /manager/Index ${session.getAttribute("locale")} 5 https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:37561 Thu 18 Feb 2021 11:35:20 AEDT ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:37560 Thu 18 Feb 2021 11:00:21 AEDT ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:37555 TGFBR2 encoding TGFB receptor 2. Conversely, TGFB1 suppressed IFNG receptor 1 and 2 genes IFNGR1 and IFNGR2. These data identify IFNG as a potent inhibitor of the TGFB-mediated seminal fluid interaction with relevant reproductive tract epithelia in mice and human. These findings raise the prospect that IFNG in the male partner's seminal fluid impairs immune adaptation for pregnancy following coitus in women.]]> Thu 18 Feb 2021 09:50:20 AEDT ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:10399 Sat 24 Mar 2018 08:08:45 AEDT ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:41 Sat 24 Mar 2018 07:42:12 AEDT ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:22111 Ifn-ε–deficient mice had increased susceptibility to infection of the FRT by the common sexually transmitted infections (STIs) herpes simplex virus 2 and Chlamydia muridarum. Thus, IFN-ε is a potent antipathogen and immunoregulatory cytokine that may be important in combating STIs that represent a major global health and socioeconomic burden.]]> Sat 24 Mar 2018 07:13:18 AEDT ]]>